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Job Board > Post-doctoral Fellow - University of Saskatchewan

Post-doctoral Fellow - University of Saskatchewan

Province: Saskatoon, Saskatchewan
Position: Post-doctoral Fellow
Deadline: March 31, 2019
Posted: February 6, 2019

Job Description / Duties

The successful applicant will be hard-working collegial individual with a strong background in immunology and a deep interest in translational research.
Applicants must have a PhD degree with 0-2 years’ experience as a post-doctoral fellow, and a strong track record of publication in peer-reviewed journals and presentations to scientific audiences. Candidates should be able to independently design and assess their own experiments and research program, but also to share projects with others in the lab and with external collaborators.

Qualifications / Required Skills

Expertise in immunology and immunologic techniques, especially flow cytometry, cell culture and model systems.
Strong ability to manage a research project and work independently, but also in a collegial fashion with others.
Strong oral and written communication and critical thinking skills.

Additional Information

Qualified candidates should submit their application by email in .pdf document that includes:
(1) a cover letter stating the candidate’s suitability for the position;
(2) a full CV, including all publications and presentations;
(3) a brief statement of research experience and interests; and
(4) contact information for three referees familiar with their research

Contact Information

Dr. John R Gordon
Division of Respirology, Critical Care and Sleep Medicine
Department of Medicine
Rm 2D30.8 (Box 3) Health Sciences Building
107 Wiggins Rd
Saskatoon, SK S7N 5E5
email: john.gordon@usask.ca

Employer Profile

The Gordon lab is a highly collaborative and collegial group within the College of Medicine at the University of Saskatchewan. We are seeking an exceptional post-doctoral fellow to lead a study on translation of regulatory dendritic cell (DCreg) therapeutic approaches for immunologic diseases. We have shown that DCreg therapy can reverse the disease phenotype in mouse models of asthma and peanut allergies, and that analogous human DCreg can suppress Th2 responses of asthmatic donors, converting these T cells into Treg. We are expanding our repertoire of human DCreg, using molecular and immunologic approaches to characterize their activities in vitro and in vivo in humanized mouse models.